Show simple item record

dc.contributor.authorYolaş, Coşkun
dc.contributor.authorKanat, Ayhan
dc.contributor.authorAydın, Mehmet Dumlu
dc.contributor.authorTürkmenoğlu, Osman Nuri
dc.contributor.authorGündoğdu, Cemal
dc.date.accessioned2020-12-19T20:03:28Z
dc.date.available2020-12-19T20:03:28Z
dc.date.issued2014
dc.identifier.citationYolas, C., Kanat, A., Aydin, M.D., Turkmenoglu, O.N., Gundogdu, C. (2014). Important casual association of carotid body and glossopharyngeal nerve and lung following experimental subarachnoid hemorrhage in rabbits. First report. Journal of the Neurological Sciences, 336(1-2), 220-226. https://doi.org/10.1016/j.jns.2013.10.047en_US
dc.identifier.issn0022-510X
dc.identifier.issn1878-5883
dc.identifier.urihttps://doi.org/10.1016/j.jns.2013.10.047
dc.identifier.urihttps://hdl.handle.net/11436/3176
dc.descriptionGundogdu, Cemal/0000-0003-2857-923X; Kanat, Ayhan/0000-0002-8189-2877en_US
dc.descriptionWOS: 000331025400038en_US
dc.descriptionPubMed: 24267741en_US
dc.description.abstractObject: the glossopharyngeal nerves (GPNs) and carotid bodies (CBs) have an important role in the continuation of the cerebral autoregulation and cardiorespiratory functions. the relationship between degenerative injury of CB and the GPN in subarachnoid hemorrhage (SAH) was studied. Methods: Twenty rabbits were included in this study. Five of them (n = 5) were used as control group. the remaining animals (n = 15) were exposed to experimental SAH. in the six animals of the SAH group, severe signs of illness were observed, and these six animals were killed in the first week after SAH. Others animals (n = 9) were followed for 20 days and then sacrificed. GPNs and CBs were examined and, the live and degenerated GPN axon number, and of CB neuron numbers were stereologically estimated. Results: the mean number of live neurons in CBs was 4206.67 +/- 148.35 and live axons of GPNs were 1211.66 +/- 14.29 in the animals of the control group. the number of degenerated neurons of CBs was 2065 +/- 110.27 and the number of degenerated axons of GPNs was 530.83 +/- 43.48 in early killed animals with SAH. the number of degenerated neurons of CBs and the number of degenerated axons of GPNs were found as 1013.89 +/- 4184 and 2270.5 +/- 13438 in the living animals with SAH, respectively. Conclusions: High number of degenerated axons of GPN and neurons of CBs of the early killed animals suggest that the mortality in early SAH might be due to GPNs injury secondary to compression of their axons or supplying vessels by the probably herniated brainstem, and secondary denervation injury of CBs, and lung. Crown Copyright (c) 2013 Published by Elsevier B.V. All rights reserved.en_US
dc.language.isoengen_US
dc.publisherElsevier Science Bven_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectSubarachnoid hemorrhageen_US
dc.subjectGlossopharyngeal nerveen_US
dc.subjectCarotid bodyen_US
dc.subjectBrain stem compressionen_US
dc.subjectCardiorespiratory arresten_US
dc.subjectDegenerative injuryen_US
dc.titleImportant casual association of carotid body and glossopharyngeal nerve and lung following experimental subarachnoid hemorrhage in rabbits. First reporten_US
dc.typearticleen_US
dc.contributor.departmentRTEÜ, Tıp Fakültesi, Cerrahi Tıp Bilimleri Bölümüen_US
dc.contributor.institutionauthorKanat, Ayhan
dc.identifier.doi10.1016/j.jns.2013.10.047
dc.identifier.volume336en_US
dc.identifier.issue01.Feben_US
dc.identifier.startpage220en_US
dc.identifier.endpage226en_US
dc.relation.journalJournal of the Neurological Sciencesen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US


Files in this item

Thumbnail

This item appears in the following Collection(s)

Show simple item record